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Therapeutic approaches for the treatment of HD and other CAG repeat... | Download Scientific Diagram
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PolyQ-independent toxicity associated with novel translational products from CAG repeat expansions | bioRxiv
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Molecular Genesis Huntington's Disease Illustration Expansion Cag Trinucleotide Sequence Htt Stock Photo by ©katerynakon 474065062
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Uninterrupted CAG repeat drives striatum-selective transcriptionopathy and nuclear pathogenesis in human Huntingtin BAC mice - ScienceDirect
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A CAG repeat-targeting artificial miRNA lowers the mutant huntingtin level in the YAC128 model of Huntington's disease: Molecular Therapy - Nucleic Acids
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Discovery of a potent small molecule inhibiting Huntington's disease (HD) pathogenesis via targeting CAG repeats RNA and Poly Q protein | Scientific Reports
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Suppression of Mutant Protein Expression in SCA3 and SCA1 Mice Using a CAG Repeat-Targeting Antisense Oligonucleotide: Molecular Therapy - Nucleic Acids
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Selective suppression of polyglutamine-expanded protein by lipid nanoparticle-delivered siRNA targeting CAG expansions in the mouse CNS: Molecular Therapy - Nucleic Acids
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Molecular Genesis Of Huntington's Disease, 3D Illustration. Expansion Of The CAG Trinucleotide Sequence In The Htt Gene Causes Production Of Mutated Huntingtin Protein Leading To Neurodegeneration Stock Photo, Picture And Royalty Free
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Molecular genesis of Huntington's disease, illustration. Expansion of the CAG trinucleotide sequence in the htt gene causes production of mutated Huntingtin protein leading to neurodegeneration, atrophy of brain basal ganglia, involuntary movements
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